Friday, September 24, 2010

BSE Surveillance Continues to Benefit Canadian Cattle Producers September 24, 2010 - Notice to Industry

Notice to Industry - BSE Surveillance Continues to Benefit Canadian Cattle Producers September 24, 2010: The Canadian Food Inspection Agency (CFIA) reminds cattle producers to continue to present eligible animals for testing under the national bovine spongiform encephalopathy (BSE) surveillance program.

The national BSE surveillance program is critical for maintaining domestic and international confidence in Canadian beef products. It continues to demonstrate the low level of BSE in Canada and our collective commitment to meeting international obligations. The program plays an important role in Canada's strategy to manage BSE and to assess the effectiveness of our control measures.

The CFIA is looking to test animals most at risk for the disease, which include

•cattle over the age of 30 months that are dead, down, dying or diseased •cattle exhibiting clinical signs of BSE Please contact the nearest CFIA office or a veterinarian to make arrangements for sampling. The CFIA provides financial support to offset the costs of veterinary examinations and carcass disposal (on-farm disposal or deadstock collection).

In Alberta, BSE surveillance is jointly managed by the Province of Alberta and the CFIA, and the program requirements are slightly different. For more information on the Canada-Alberta BSE Surveillance Program, including details on the eligibility criteria of the reimbursement program, contact the Province of Alberta.

Continued stewardship and vigilance from cattle producers is critical to the success of the BSE surveillance program, and ultimately to the control of the disease in Canada.

For more information on the national BSE surveillance program, call 1-800-442-2342 or visit www.inspection.gc.ca.

http://www.inspection.gc.ca/english/anima/heasan/disemala/bseesb/ind20100924e.shtml



Since Canada has taken over some of the USDA et al's bad habits of not reporting BSE cases, until the media finds out, I only hope they have not taken up the bad habit of only testing healthy cattle brains for the BSE surveillance, cattle brains they knew did not have BSE..............



Mad Cow Disease Case Hidden For Weeks By Canadian and U.S. Agencies

HuffPost Citizen Reporting Nikki Zeichner Posted: 03-11-10 07:08 PM

On February 25, 2010, the Canadian Food Inspection Agency (CFIA) confirmed a recent case of bovine spongiform encephalopathy (BSE), commonly known as Mad Cow Disease, in a 72 month-old cow.

This case was detected through the national BSE surveillance program and was not made public on the CFIA website until March 10 -- hours after a press release was distributed by the advocacy group, Ranchers-Cattlemen Action Legal Fund, United Stockgrowers of America (RCALF USA).

"The CFIA said the BSE-positive case was confirmed Feb. 25, 2010, which means the CFIA and all other governments who knew about this latest BSE case kept it a secret from the public for almost two weeks," said R-CALF USA CEO Bill Bullard. "If we had not discovered this information, the public may never have known."

According to a CFIA spokesperson, the Agency updates its website with cases of federally reportable diseases found in farmed animals once a month. Immediate updates are made only when it determines that there are reportable, foreign, or newly emerging diseases which pose significant health or economic risks.

The USDA claims that its Animal and Plant Health Inspection Service (APHIS) determined that the risk presented here in trade in beef or cattle from Canada is negligible.

According to USDA spokesperson, Caleb Weaver, "APHIS followed international standards, as defined by the World Organization for Animal Health (OIE), in making this determination."

Both the Canadian Food Inspection Agency and the USDA admit that there is no way to test live animals for BSE; testing for BSE can only be done by postmortem microscopic examination of the animal's brain tissue. These tests are conducted on dead animals to detect the possibility of infection within a specific cattle population. BSE testing in Canada is voluntary and, according to Canadian Food Inspection Agency data, rates of BSE testing of cattle being exported to the U.S. are on the decline.

Recent USDA regulations permit live Canadian cattle born after March 1, 1999, to be imported into the United States without mandatory BSE testing. This means that the infected cow would have been eligible for import into the U.S. cattle market had it been alive.

http://www.huffingtonpost.com/2010/03/11/mad-cow-disease-case-hidd_n_494881.html



U.S. Department of State

Cases Regarding the Border Closure due to BSE Concerns

Several Canadian claimants have submitted notices of arbitration under the UNCITRAL Arbitration Rules alleging that the United States has violated NAFTA Chapter Eleven by closing the border to the importation of Canadian cattle after the discovery in 2003 of a case of bovine spongiform encephalopathy (BSE or mad cow disease) in a cow in Alberta, Canada. Claimants are Canadian citizens and corporations that own and operate cattle feeding, feedlot and transportation businesses in Canada, which they allege were damaged by the border closure.

Claimants allege that the border closure violates NAFTA Article 1102 (national treatment). The notices of arbitration seek damages of varying amounts, ranging from CAN$38,000 to CAN$95 million. The total amount of damages sought by claimants is approximately US$235 million.

On January 28, 2008, the tribunal issued its Award on Jurisdiction, dismissing the claims against the United States in their entirety. The tribunal’s award, and other documents in the case, appear below.

-01/28/08 Award on Jurisdiction [575 Kb] -10/10/07 Transcript of the Hearing on the Preliminary Issue - Day Two [166 Kb] -10/09/07 Transcript of the Hearing on the Preliminary Issue - Day One [260 Kb] -08/03/07 Procedural Order No. 3 [26 Kb] -07/05/07 Claimants' Rejoinder on the Preliminary Issue [849 Kb] -05/01/07 U.S. Reply on the Preliminary Issue [245 Kb] -03/01/07 Article 1128 Submission of Mexico [47 Kb] -01/30/07 Claimants' Response on the Preliminary Question [2303 Kb] -12/01/06 U.S. Memorial on the Preliminary Issue [167 Kb] -11/07/06 Procedural Order No. 2 [62 Kb] -10/20/06 Procedural Order No. 1 [122 Kb] -06/02/05 Jim McNall Notice of Arbitration [393 Kb] -06/02/05 Leslie Smith Notice of Arbitration [393 Kb] -06/02/05 Michael Sears Notice of Arbitration [393 Kb] -06/02/05 Rex Vandenberg Notice of Arbitration [393 Kb] -06/02/05 Richard Hiebert Notice of Arbitration [392 Kb] -06/02/05 Rod Oosterbroek Notice of Arbitration [392 Kb] -06/02/05 TER Cattle Notice of Arbitration [393 Kb] -05/20/05 Andrew Oosterbroek Notice of Arbitration [392 Kb] -05/20/05 Brad Hopkins Notice of Arbitration [396 Kb] -05/20/05 Brent Byers Notice of Arbitration [395 Kb] -05/20/05 Brent Fisher Notice of Arbitration [393 Kb] -05/20/05 Byron Sedore Notice of Arbitration [394 Kb] -05/20/05 Chris Irwin Notice of Arbitration [394 Kb] -05/20/05 Cornelius Van Hal Notice of Arbitration [392 Kb] -05/20/05 Darren Johnston Notice of Arbitration [394 Kb] -05/20/05 Dave Knapp Notice of Arbitration [394 Kb] -05/20/05 David Hewitt Notice of Arbitration [394 Kb] -05/20/05 Donald Procter Notice of Arbitration [394 Kb] -05/20/05 George Adams Notice of Arbitration [392 Kb] -05/20/05 Glen Thompson Notice of Arbitration [392 Kb] -05/20/05 Graham Alexander Notice of Arbitration [394 Kb] -05/20/05 Helmut Friesen Notice of Arbitration [393 Kb] -05/20/05 James Wiskerke Notice of Arbitration [392 Kb] -05/20/05 Joseph Daunt Notice of Arbitration [394 Kb] -05/20/05 Keith Kerr Notice of Arbitration [394 Kb] -05/20/05 Ken Andreychuk Notice of Arbitration [396 Kb] -05/20/05 Kevin Freiburger Notice of Arbitration [394 Kb] -05/20/05 Larry Brodersen Notice of Arbitration [392 Kb] -05/20/05 Lee Robson Notice of Arbitraiton [450 Kb] -05/20/05 Maria Vanden Elzen Notice of Arbitration [413 Kb] -05/20/05 Murray Johnston Notice of Arbitration [395 Kb] -05/20/05 NFL Holdings Notice of Arbitration [456 Kb] -05/20/05 Paul Gowing Notice of Arbitration [394 Kb] -05/20/05 Paul MacIntyre Notice of Arbitration [394 Kb] -05/20/05 Peter Schwenk Notice of Arbitration [448 Kb] -05/20/05 Peter Vander Heyden Notice of Arbitration [415 Kb] -05/20/05 Robert Emerson Notice of Arbitration [394 Kb] -05/20/05 Robert Laidlaw Notice of Arbitration [393 Kb] -05/20/05 Ron Coulter Notice of Arbitration [393 Kb] -05/20/05 Ross McCall Notice of Arbitration [394 Kb] -05/20/05 Ryan Kasko Notice of Arbitration [393 Kb] -05/11/05 Barry Hillman Notice of Arbitration [392 Kb] -05/11/05 Ben Gardiner Notice of Arbitration [416 Kb] -05/11/05 Bernie Loman Notice of Arbitration [392 Kb] -05/11/05 Blair Bieman Notice of Arbitration [394 Kb] -05/11/05 Blake Holtman Notice of Arbitration [393 Kb] -05/11/05 Bruce Groenenboom Notice of Arbitration [403 Kb] -05/11/05 Butch Martin Notice of Arbitration [441 Kb] -05/11/05 Dale Pallister Notice of Arbitration [394 Kb] -05/11/05 Darwin Ullery Notice of Arbitration [412 Kb] -05/11/05 Dave Gardiner Notice of Arbitration [415 Kb] -05/11/05 Dave Johnston Notice of Arbitration [415 Kb] -05/11/05 Dave Matthies, Notice of Arbitration [421 Kb] -05/11/05 David Millsap Notice of Arbitration [394 Kb] -05/11/05 Doug Briggs Notice of Arbitration [394 Kb] -05/11/05 Doug Nieboer Notice of Arbitration [392 Kb] -05/11/05 Doug Shelswel Notice of Arbitration [394 Kb] -05/11/05 Ed Stronks Notice of Arbitration [413 Kb] -05/11/05 Eric Thacker Notice of Arbitration [394 Kb] -05/11/05 Eve t Kraayenbrink Notice of Arbitration [415 Kb] -05/11/05 Firmin Declercq Notice of Arbitration [392 Kb] -05/11/05 Frank Zettler Notice of Arbitration [395 Kb] -05/11/05 G. Lee Hochstein Notice of Arbitration [374 Kb] -05/11/05 George Alton Notice of Arbitration [394 Kb] -05/11/05 George Maxwell Notice of Arbitration [394 Kb] -05/11/05 Glen Armitage Notice of Arbitration [392 Kb] -05/11/05 Grant Nelson Notice of Arbitration [393 Kb] -05/11/05 Harry Duban Notice of Arbitration [393 Kb] -05/11/05 Harry Vandersteen Notice of Arbitration [393 Kb] -05/11/05 Harry Welsch Notice of Arbitration [393 Kb] -05/11/05 Henry Van Hall Notice of Arbitration [392 Kb] -05/11/05 Herb Groenenboom Notice of Arbitration [392 Kb] -05/11/05 Herbert Serfas Notice of Arbitration [403 Kb] -05/11/05 Herman Stroeve Notice of Arbitration [449 Kb] -05/11/05 Ian MacLean Notice of Arbitration [394 Kb] -05/11/05 Jim Steed Notice of Arbitration [394 Kb] -05/11/05 Joe Stroeve Notice of Arbitration [423 Kb] -05/11/05 John Schooten Notice of Arbitration [392 Kb] -05/11/05 John Stroeve Notice of Arbitration [423 Kb] -05/11/05 John Vander Heyden Notice of Arbitration [392 Kb] -05/11/05 Julie Coe Notice of Arbitration [394 Kb] -05/11/05 Keith Scott Notice of Arbitration [392 Kb] -05/11/05 Larry Lehrbass Notice of Arbitration [394 Kb] -05/11/05 Leighton Kolk Notice of Arbitration [392 Kb] -05/11/05 Lloyd Sproule Notice of Arbitration [391 Kb] -05/11/05 Louis Ypma Notice of Arbitration [392 Kb] -05/11/05 Marty Wren Notice of Arbitration [392 Kb] -05/11/05 Mary Conlin Notice of Arbitration [395 Kb] -05/11/05 Murray Brodhagen Notice of Arbitration [394 Kb] -05/11/05 Nick Popovic Notice of Arbitration [394 Kb] -05/11/05 Paul Adams Notice of Arbitration [392 Kb] -05/11/05 Renus Van Hal Notice of Arbitration [392 Kb] -05/11/05 Richard Visser Notice of Arbitration [392 Kb] -05/11/05 Rients Wever Notice of Arbitration [392 Kb] -05/11/05 Robert Cooke Notice of Arbitration [415 Kb] -05/11/05 Robert Vander Heyden Notice of Arbitration [393 Kb] -05/11/05 Ryan Gibson Notice of Arbitration [392 Kb] -05/11/05 Steve McKague Notice of Arbitration [394 Kb] -05/11/05 Stuart Alton Notice of Arbitration [394 Kb] -05/11/05 Ward Takeda Notice of Arbitration [393 Kb] -05/11/05 Wayne Beattie Notice of Arbitration [394 Kb] -05/11/05 Wilfred Haines Notice of Arbitration [415 Kb] -03/16/05 Cor Van Raay Notice of Arbitration [396 Kb] -03/16/05 Joe Groenenboom Notice of Arbitration [392 Kb] -03/16/05 John Vander Heyden Notice of Arbitration [393 Kb] -03/16/05 Larry Nolan Notice of Arbitration [393 Kb] -03/16/05 Theodorus de Boer Notice of Arbitration [392 Kb]

http://www.state.gov/s/l/c14683.htm



P.9.21

Molecular characterization of BSE in Canada

Jianmin Yang1, Sandor Dudas2, Catherine Graham2, Markus Czub3, Tim McAllister1, Stefanie Czub1 1Agriculture and Agri-Food Canada Research Centre, Canada; 2National and OIE BSE Reference Laboratory, Canada; 3University of Calgary, Canada

Background: Three BSE types (classical and two atypical) have been identified on the basis of molecular characteristics of the misfolded protein associated with the disease. To date, each of these three types have been detected in Canadian cattle.

Objectives: This study was conducted to further characterize the 16 Canadian BSE cases based on the biochemical properties of there associated PrPres. Methods: Immuno-reactivity, molecular weight, glycoform profiles and relative proteinase K sensitivity of the PrPres from each of the 16 confirmed Canadian BSE cases was determined using modified Western blot analysis.

Results: Fourteen of the 16 Canadian BSE cases were C type, 1 was H type and 1 was L type. The Canadian H and L-type BSE cases exhibited size shifts and changes in glycosylation similar to other atypical BSE cases. PK digestion under mild and stringent conditions revealed a reduced protease resistance of the atypical cases compared to the C-type cases. N terminal- specific antibodies bound to PrPres from H type but not from C or L type. The C-terminal-specific antibodies resulted in a shift in the glycoform profile and detected a fourth band in the Canadian H-type BSE.

Discussion: The C, L and H type BSE cases in Canada exhibit molecular characteristics similar to those described for classical and atypical BSE cases from Europe and Japan. This supports the theory that the importation of BSE contaminated feedstuff is the source of C-type BSE in Canada. It also suggests a similar cause or source for atypical BSE in these countries.

http://www.prion2009.com/sites/default/files/Prion2009_Book_of_Abstracts.pdf



Wednesday, August 11, 2010

REPORT ON THE INVESTIGATION OF THE SIXTEENTH CASE OF BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN CANADA

http://bse-atypical.blogspot.com/2010/08/report-on-investigation-of-sixteenth.html



Thursday, August 19, 2010

REPORT ON THE INVESTIGATION OF THE SEVENTEENTH CASE OF BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN CANADA

http://bseusa.blogspot.com/2010/08/report-on-investigation-of-seventeenth.html



Monday, August 30, 2010

Bovine Spongiform Encephalopathy (BSE) CANADA Import Policy for Bovine Animals and Their Products (TAHD-DSAT-IE-2005-9-2) Import Policy updates August

http://madcowtesting.blogspot.com/2010/08/bovine-spongiform-encephalopathy-bse.html



Thursday, August 19, 2010

SCRAPIE CANADA UPDATE Current as of 2010-07-31 The following table lists sheep flocks and/or goat herds confirmed to be infected with scrapie in Canada in 2010.

Current as of: 2010-07-31

http://nor-98.blogspot.com/2010/08/scrapie-canada-update-current-as-of.html



Wednesday, August 18, 2010

Incidence of CJD Deaths Reported by CJD-SS in Canada as of July 31, 2010

http://creutzfeldt-jakob-disease.blogspot.com/2010/08/incidence-of-cjd-deaths-reported-by-cjd.html



Monday, August 9, 2010

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)

(please watch and listen to the video and the scientist speaking about atypical BSE and sporadic CJD and listen to Professor Aguzzi)

http://prionunitusaupdate2008.blogspot.com/2010/08/national-prion-disease-pathology.html



Subject: USDA OIG SEMIANNUAL REPORT TO CONGRESS FY 2007 1st Half (bogus BSE sampling FROM HEALTHY USDA CATTLE) Date: June 21, 2007 at 2:49 pm PST

Owner and Corporation Plead Guilty to Defrauding Bovine Spongiform Encephalopathy (BSE) Surveillance Program

An Arizona meat processing company and its owner pled guilty in February 2007 to charges of theft of Government funds, mail fraud, and wire fraud. The owner and his company defrauded the BSE Surveillance Program when they falsified BSE Surveillance Data Collection Forms and then submitted payment requests to USDA for the services. In addition to the targeted sample population (those cattle that were more than 30 months old or had other risk factors for BSE), the owner submitted to USDA, or caused to be submitted, BSE obex (brain stem) samples from healthy USDA-inspected cattle. As a result, the owner fraudulently received approximately $390,000. Sentencing is scheduled for May 2007.

snip...

Topics that will be covered in ongoing or planned reviews under Goal 1 include:

soundness of BSE maintenance sampling (APHIS),

implementation of Performance-Based Inspection System enhancements for specified risk material (SRM) violations and improved inspection controls over SRMs (FSIS and APHIS),

snip...

The findings and recommendations from these efforts will be covered in future semiannual reports as the relevant audits and investigations are completed.

4 USDA OIG SEMIANNUAL REPORT TO CONGRESS FY 2007 1st Half

http://www.usda.gov/oig/webdocs/sarc070619.pdf



THE USDA JUNE 2004 ENHANCED BSE SURVEILLANCE PROGRAM WAS TERRIBLY FLAWED ;

CDC DR. PAUL BROWN TSE EXPERT COMMENTS 2006

In an article today for United Press International, science reporter Steve Mitchell writes:

Analysis: What that mad cow means

By STEVE MITCHELL UPI Senior Medical Correspondent

WASHINGTON, March 15 (UPI) -- The U.S. Department of Agriculture was quick to assure the public earlier this week that the third case of mad cow disease did not pose a risk to them, but what federal officials have not acknowledged is that this latest case indicates the deadly disease has been circulating in U.S. herds for at least a decade.

The second case, which was detected last year in a Texas cow and which USDA officials were reluctant to verify, was approximately 12 years old.

These two cases (the latest was detected in an Alabama cow) present a picture of the disease having been here for 10 years or so, since it is thought that cows usually contract the disease from contaminated feed they consume as calves. The concern is that humans can contract a fatal, incurable, brain-wasting illness from consuming beef products contaminated with the mad cow pathogen.

"The fact the Texas cow showed up fairly clearly implied the existence of other undetected cases," Dr. Paul Brown, former medical director of the National Institutes of Health's Laboratory for Central Nervous System Studies and an expert on mad cow-like diseases, told United Press International. "The question was, 'How many?' and we still can't answer that."

Brown, who is preparing a scientific paper based on the latest two mad cow cases to estimate the maximum number of infected cows that occurred in the United States, said he has "absolutely no confidence in USDA tests before one year ago" because of the agency's reluctance to retest the Texas cow that initially tested positive.

USDA officials finally retested the cow and confirmed it was infected seven months later, but only at the insistence of the agency's inspector general.

"Everything they did on the Texas cow makes everything they did before 2005 suspect," Brown said.

Despite this, Brown said the U.S. prevalence of mad cow, formally known as bovine spongiform encephalopathy, or BSE, did not significantly threaten human or cattle health.

"Overall, my view is BSE is highly unlikely to pose any important risk either in cattle feed or human feed," he said.

However, Jean Halloran of Consumers Union in Yonkers, N.Y., said consumers should be troubled by the USDA's secrecy and its apparent plan to dramatically cut back the number of mad cow tests it conducts.

"Consumers should be very concerned about how little we know about the USDA's surveillance program and the failure of the USDA to reveal really important details," Halloran told UPI. "Consumers have to be really concerned if they're going to cut back the program," she added.

Last year the USDA tested more than 300,000 animals for the disease, but it has proposed, even in light of a third case, scaling back the program to 40,000 tests annually.

"They seem to be, in terms of actions and policies, taking a lot more seriously the concerns of the cattle industry than the concerns of consumers," Halloran said. "It's really hard to know what it takes to get this administration to take action to protect the public."

The USDA has insisted that the safeguards of a ban on incorporating cow tissue into cattle feed (which is thought to spread the disease) and removal of the most infectious parts of cows, such as the brain and spinal cord, protect consumers. But the agency glosses over the fact that both of these systems have been revealed to be inadequately implemented.

The feed ban, which is enforced by the Food and Drug Administration, has been criticized by the Government Accountability Office in two reports, the most recent coming just last year. The GAO said the FDA's enforcement of the ban continues to have weaknesses that "undermine the nation's firewall against BSE."

USDA documents released last year showed more than 1,000 violations of the regulations requiring the removal of brains and spinal cords in at least 35 states, Puerto Rico and the Virgin Islands, with some plants being cited repeatedly for infractions. In addition, a violation of similar regulations that apply to beef exported to Japan is the reason why Japan closed its borders to U.S. beef in January six weeks after reopening them.

Other experts also question the adequacy of the USDA's surveillance system. The USDA insists the prevalence of mad cow disease is low, but the agency has provided few details of its surveillance program, making it difficult for outside experts to know if the agency's monitoring plan is sufficient.

"It's impossible to judge the adequacy of the surveillance system without having a breakdown of the tested population by age and risk status," Elizabeth Mumford, a veterinarian and BSE expert at Safe Food Solutions in Bern, Switzerland, a company that provides advice on reducing mad cow risk to industry and governments, told UPI.

"Everybody would be happier and more confident and in a sense it might be able to go away a little bit for (the USDA) if they would just publish a breakdown on the tests," Mumford added.

UPI requested detailed records about animals tested under the USDA's surveillance plan via the Freedom of Information Act in May 2004 but nearly two years later has not received any corresponding documents from the agency, despite a federal law requiring agencies to comply within 30 days. This leaves open the question of whether the USDA is withholding the information, does not have the information or is so haphazardly organized that it cannot locate it.

Mumford said the prevalence of the disease in U.S. herds is probably quite low, but there have probably been other cases that have so far gone undetected. "They're only finding a very small fraction of that low prevalence," she said.

Mumford expressed surprise at the lack of concern about the deadly disease from American consumers. "I would expect the U.S. public to be more concerned," she said.

Markus Moser, a molecular biologist and chief executive officer of Prionics, a Swiss firm that manufactures BSE test kits, told UPI one concern is that if people are infected, the mad cow pathogen could become "humanized" or more easily transmitted from person to person.

"Transmission would be much easier, through all kinds of medical procedures" and even through the blood supply, Moser said.

© Copyright 2006 United Press International, Inc. All Rights Reserved

http://www.upi.com/ConsumerHealthDaily/view.php?StoryID=20060315-055557-1284r


http://www.upi.com/Science_News/2003/12/30/Mad-Cow-Linked-to-thousands-of-CJD-cases/UPI-47861072816318/


CDC - Bovine Spongiform Encephalopathy and Variant Creutzfeldt ... Dr. Paul Brown is Senior Research Scientist in the Laboratory of Central Nervous System ... Address for correspondence: Paul Brown, Building 36, Room 4A-05, ...

http://www.cdc.gov/ncidod/eid/vol7no1/brown.htm



PAUL BROWN COMMENT TO ME ON THIS ISSUE

Tuesday, September 12, 2006 11:10 AM

"Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency." ........TSS


http://madcowtesting.blogspot.com/2009/07/mad-cow-cover-up-usa-masked-as-sporadic.html



OR, what the Honorable Phyllis Fong of the OIG found ;

Audit Report Animal and Plant Health Inspection Service Bovine Spongiform Encephalopathy (BSE) Surveillance Program ­ Phase II and Food Safety and Inspection Service

Controls Over BSE Sampling, Specified Risk Materials, and Advanced Meat Recovery Products - Phase III

Report No. 50601-10-KC January 2006

Finding 2 Inherent Challenges in Identifying and Testing High-Risk Cattle Still Remain


http://www.usda.gov/oig/webdocs/50601-10-KC.pdf



THIS is just ONE month report, of TWO recalls of prohibited banned MBM, which is illegal, mixed with 85% blood meal, which is still legal, but yet we know the TSE/BSE agent will transmit blood. we have this l-BSE in North America that is much more virulent and there is much concern with blood issue and l-BSE as there is with nvCJD in humans. some are even starting to be concerned with sporadic CJD and blood, and there are studies showing transmission there as well. ... this is one month recall page, where 10 MILLION POUNDS OF BANNED MAD COW FEED WENT OUT INTO COMMERCE, TO BE FED OUT. very little of the product that reaches commerce is ever returned via recall, very, very little. this was 2007, TEN YEARS AFTER THE AUGUST 4, 1997, PARTIAL AND VOLUNTARY MAD COW FEED BAN IN THE USA, that was nothing but ink on paper. i have listed the tonnage of mad cow feed that was in ALABAMA in one of the links too, this is where the infamous g-h-BSEalabama case was, a genetic relation matching the new sporadic CJD in the USA. seems this saga just keeps getting better and better.......$$$



10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN COMMERCE USA 2007

Date: March 21, 2007 at 2:27 pm PST

RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II

___________________________________

PRODUCT

Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007

CODE

Cattle feed delivered between 01/12/2007 and 01/26/2007

RECALLING FIRM/MANUFACTURER

Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.

Firm initiated recall is ongoing.


REASON

Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.

VOLUME OF PRODUCT IN COMMERCE

42,090 lbs.

DISTRIBUTION

WI


___________________________________


PRODUCT

Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007

CODE

The firm does not utilize a code - only shipping documentation with commodity and weights identified.

RECALLING FIRM/MANUFACTURER

Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007. Firm initiated recall is complete.


REASON

Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.


VOLUME OF PRODUCT IN COMMERCE

9,997,976 lbs.



DISTRIBUTION

ID and NV


END OF ENFORCEMENT REPORT FOR MARCH 21, 2007

http://www.fda.gov/Safety/Recalls/EnforcementReports/2007/ucm120446.htm



Wednesday, September 22, 2010

Docket APHIS-2010-0056 National Veterinary Services Laboratories; Bovine Spongiform Encephalopathy Surveillance Program Documents COMMENT SUBMISSION

Docket No. APHIS-2010-0056

http://madcowtesting.blogspot.com/2010/09/docket-aphis-2010-0056-national.html


Docket No. 03-080-1 -- USDA ISSUES PROPOSED RULE TO ALLOW LIVE ANIMAL IMPORTS FROM CANADA

https://web01.aphis.usda.gov/BSEcom.nsf/0/b78ba677e2b0c12185256dd300649f9d?OpenDocument&AutoFramed


PLEASE SEE FULL TEXT HERE ;

Docket No. 03-080-1 -- USDA ISSUES PROPOSED RULE TO ALLOW LIVE ANIMAL IMPORTS FROM CANADA

http://madcowfeed.blogspot.com/2008/07/docket-no-03-080-1-usda-issues-proposed.html


Docket APHIS-2006-0026 Docket Title Bovine Spongiform Encephalopathy; Animal Identification and Importation of Commodities Docket Type Rulemaking Document APHIS-2006-0026-0001 Document Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions, Identification of Ruminants and Processing and Importation of Commodities Public Submission APHIS-2006-0026-0012 Public Submission Title Comment from Terry S Singletary

http://www.regulations.gov/fdmspublic/component/main?main=DocumentDetail&o=09000064801e47e1


Docket APHIS-2006-0041 Docket Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions; Importation of Live Bovines and Products Derived from Bovines Commodities Docket Type Rulemaking Document APHIS-2006-0041-0001 Document Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions; Importation of Live Bovines and Products Derived From Bovines Public Submission APHIS-2006-0041-0028 Public Submission Title Comment from Terry S Singletary

Comment 2006-2007 USA AND OIE POISONING GLOBE WITH BSE MRR POLICY

THE USA is in a most unique situation, one of unknown circumstances with human and animal TSE. THE USA has the most documented TSE in different species to date, with substrains growing in those species (BSE/BASE in cattle and CWD in deer and elk, there is evidence here with different strains), and we know that sheep scrapie has over 20 strains of the typical scrapie with atypical scrapie documented and also BSE is very likely to have passed to sheep. all of which have been rendered and fed back to animals for human and animal consumption, a frightening scenario. WE do not know the outcome, and to play with human life around the globe with the very likely TSE tainted products from the USA, in my opinion is like playing Russian roulette, of long duration, with potential long and enduring consequences, of which once done, cannot be undone. These are the facts as I have come to know through daily and extensive research of TSE over 9 years, since 12/14/97. I do not pretend to have all the answers, but i do know to continue to believe in the ukbsenvcjd only theory of transmission to humans of only this one strain from only this one TSE from only this one part of the globe, will only lead to further failures, and needless exposure to humans from all strains of TSE, and possibly many more needless deaths from TSE via a multitude of proven routes and sources via many studies with primates and rodents and other species.

MY personal belief, since you ask, is that not only the Canadian border, but the USA border, and the Mexican border should be sealed up tighter than a drum for exporting there TSE tainted products, until a validated, 100% sensitive test is available, and all animals for human and animal consumption are tested. all we are doing is the exact same thing the UK did with there mad cow poisoning when they exported it all over the globe, all the while knowing what they were doing. this BSE MRR policy is nothing more than a legal tool to do just exactly what the UK did, thanks to the OIE and GW, it's legal now. and they executed Saddam for poisoning ???

go figure. ...

http://www.regulations.gov/fdmspublic/component/main?main=DocumentDetail&o=09000064801f8151


Docket APHIS-2006-0041 Docket Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions; Importation of Live Bovines and Products Derived from Bovines Commodities Docket Type Rulemaking Document APHIS-2006-0041-0001 Document Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions; Importation of Live Bovines and Products Derived From Bovines Public Submission APHIS-2006-0041-0028.1 Public Submission Title Attachment to Singletary comment

January 28, 2007

Greetings APHIS,

I would kindly like to submit the following to ;

BSE; MRR; IMPORTATION OF LIVE BOVINES AND PRODUCTS DERIVED FROM BOVINES [Docket No. APHIS-2006-0041] RIN 0579-AC01

http://www.regulations.gov/fdmspublic/ContentViewer?objectId=09000064801f8152&disposition=attachment&contentType=msw8



TSS

Wednesday, September 22, 2010

Docket APHIS-2010-0056 National Veterinary Services Laboratories; Bovine Spongiform Encephalopathy Surveillance Program Documents COMMENT SUBMISSION

Docket No. APHIS-2010-0056



[Federal Register: September 22, 2010 (Volume 75, Number 183)] [Notices] [Page 57737-57738] From the Federal Register Online via GPO Access [wais.access.gpo.gov] [DOCID:fr22se10-22]

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DEPARTMENT OF AGRICULTURE

Animal and Plant Health Inspection Service

[Docket No. APHIS-2010-0056]

Notice of Request for Approval of an Information Collection; National Veterinary Services Laboratories; Bovine Spongiform Encephalopathy Surveillance Program Documents

AGENCY: Animal and Plant Health Inspection Service, USDA.

ACTION: New information collection; comment request.

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SUMMARY: In accordance with the Paperwork Reduction Act of 1995, this notice announces the Animal and Plant Health Inspection Service's intention to request approval of an information collection associated with National Veterinary Services Laboratories diagnostic support for the bovine spongiform encephalopathy surveillance program.

DATES: We will consider all comments that we receive on or before November 22, 2010.

ADDRESSES: You may submit comments by either of the following methods:

Federal eRulemaking Portal: Go to

http://www.regulations.gov/fdmspublic/component/main?main=DocketDetail&d=APHIS-2010-0056


to submit or view comments and to view supporting and related materials available electronically.

Postal Mail/Commercial Delivery: Please send one copy of your comment to Docket No. APHIS-2010-0056, Regulatory Analysis and Development, PPD, APHIS, Station 3A-03.8, 4700 River Road Unit 118, Riverdale, MD 20737-1238. Please state that your comment refers to Docket No. APHIS-2010-0056.

Reading Room: You may read any comments that we receive on this docket in our reading room. The reading room is located in room 1141 of the USDA South Building, 14th Street and Independence Avenue SW., Washington, DC. Normal reading room hours are 8 a.m. to 4:30 p.m., Monday through Friday, except holidays. To be sure someone is there to help you, please call (202) 690-2817 before coming.

Other Information: Additional information about APHIS and its programs is available on the Internet at (http://www.aphis.usda.gov).


FOR FURTHER INFORMATION CONTACT: For information on documents associated with the bovine spongiform encephalopathy surveillance program, contact Dr. Nancy Clough, VS, APHIS, 1920 Dayton Avenue, Ames, IA 50010; (515) 337-7989. For copies of more detailed information on the information collection, contact Mrs. Celeste Sickles, APHIS' Information Collection Coordinator, at (301) 851-2908.

SUPPLEMENTARY INFORMATION:

Title: National Veterinary Services Laboratories; Bovine Spongiform Encephalopathy Surveillance Program Documents.

OMB Number: 0579-xxxx.

Type of Request: Approval of an information collection.

Abstract: Under the Animal Health Protection Act (7 U.S.C. 8301 et seq.), the Animal and Plant Health Inspection Service (APHIS) of the U.S. Department of Agriculture (USDA) is authorized, among other things, to carry out activities to detect, control, and eradicate pests and diseases of livestock within the United States. APHIS' National Veterinary Services Laboratories (NVSL) safeguard U.S. animal health and contribute to public health by ensuring that timely and accurate laboratory support is provided by their nationwide animal health diagnostic system.

NVSL conducts an ongoing surveillance program to monitor and assess changes to the BSE status of U.S cattle and to provide mechanisms for early detection of BSE. NVSL collects samples from a variety of sites and from the cattle populations where the disease is most likely to be detected. These diagnostic services involve information collection activities, including the BSE Surveillance Submission Form/Continuation Sheet (VS Forms 17-146/17-146A) and the BSE Surveillance Data Collection Form (VS Form 17-131).

We are asking the Office of Management and Budget (OMB) to approve our use of these information collection activities for 3 years.

The purpose of this notice is to solicit comments from the public (as well as agencies) concerning our information collection. These comments will help us:

(1) Evaluate whether the collection of information is necessary for the proper performance of the functions of the Agency, including whether the information will have practical utility;

(2) Evaluate the accuracy of our estimate of the burden of the information collection, including the validity of the methodology and assumptions used;

(3) Enhance the quality, utility, and clarity of the information to be collected; and

(4) Minimize the burden of the information collection on those who are to respond, through use, as appropriate, of automated, electronic, mechanical, and other collection technologies, e.g., permitting electronic submission of responses.

Estimate of burden: The public reporting burden for this collection of information is estimated to average 0.1000068 hours per response.

Respondents: Slaughter establishments, offsite collection facilities for condemned slaughter, 3D/4D facilities, veterinary diagnostic laboratories, and State animal health personnel.

Estimated annual number of respondents: 60.

Estimated annual number of responses per respondent: 732.95.

Estimated annual number of responses: 43,977.

Estimated total annual burden on respondents: 4,398 hours. (Due to averaging, the total annual burden hours may not equal the product of the annual number of responses multiplied by the reporting burden per response.)

All responses to this notice will be summarized and included in the request for OMB approval. All comments will also become a matter of public record.

[[Page 57738]]

Done in Washington, DC, this 16\th\ day of September 2010.

Kevin Shea

Acting Administrator, Animal and Plant Health Inspection Service. [FR Doc. 2010-23670 Filed 9-21-10; 12:14 pm]

BILLING CODE 3410-34-S


http://edocket.access.gpo.gov/2010/2010-23670.htm



http://edocket.access.gpo.gov/2010/pdf/2010-23670.pdf






National Veterinary Services Laboratories; Bovine Spongiform Encephalopathy Surveillance Program Documents


Docket No. APHIS-2010-0056 COMMENT SUBMISSION



Greetings APHIS et al, I wish to submit the following ;


NO amount of reduction of paperwork will help or hurt the surveillance of Bovine Spongiform Encephalopathy, because the 2004 Enhanced BSE testing and surveillance of BSE consisted of nothing less than testing perfectly healthy USDA certified cow brains. OR the fact that in Texas, the SSS policy of shoot, shovel, and shut the hell up has and still is in progress. OR, when you have a stumbling and staggering highly suspect BSE case, just send this highly suspect BSE cow straight to be rendered. OR feeding dead stock downers for 4 plus years to our school children all across the Nation (the most high risk cattle for BSE). The fact the USA has been feeding cows to cows since the partial and voluntary feed ban was put in place, this is all a waste of time. The mad cows are and have been out of the barn, humans are dying from it, and it is being mask as sporadic CJD of "unknown phenotype", and sporadic CJD is rising. I remember once back in or around 2001 or 2002, it was an FDA warning letter about banned ruminant feed and or mislabeling there from, but what stunned me the most was the fact the record keeping for this ranchers feeding practices was kept on a board in the barn with crayons. that was it. I only hope that this type record keeping is not where we are headed back too. ...

MY point, what is the point of this charade anyway ???

Subject: USDA OIG SEMIANNUAL REPORT TO CONGRESS FY 2007 1st Half (bogus BSE sampling FROM HEALTHY USDA CATTLE) Date: June 21, 2007 at 2:49 pm PST

Owner and Corporation Plead Guilty to Defrauding Bovine Spongiform Encephalopathy (BSE) Surveillance Program

An Arizona meat processing company and its owner pled guilty in February 2007 to charges of theft of Government funds, mail fraud, and wire fraud. The owner and his company defrauded the BSE Surveillance Program when they falsified BSE Surveillance Data Collection Forms and then submitted payment requests to USDA for the services. In addition to the targeted sample population (those cattle that were more than 30 months old or had other risk factors for BSE), the owner submitted to USDA, or caused to be submitted, BSE obex (brain stem) samples from healthy USDA-inspected cattle. As a result, the owner fraudulently received approximately $390,000. Sentencing is scheduled for May 2007.

snip...

Topics that will be covered in ongoing or planned reviews under Goal 1 include:

soundness of BSE maintenance sampling (APHIS),

implementation of Performance-Based Inspection System enhancements for specified risk material (SRM) violations and improved inspection controls over SRMs (FSIS and APHIS),

snip...

The findings and recommendations from these efforts will be covered in future semiannual reports as the relevant audits and investigations are completed.

4 USDA OIG SEMIANNUAL REPORT TO CONGRESS FY 2007 1st Half

http://www.usda.gov/oig/webdocs/sarc070619.pdf



THE USDA JUNE 2004 ENHANCED BSE SURVEILLANCE PROGRAM WAS TERRIBLY FLAWED ;

CDC DR. PAUL BROWN TSE EXPERT COMMENTS 2006

In an article today for United Press International, science reporter Steve Mitchell writes:


Analysis: What that mad cow means

By STEVE MITCHELL UPI Senior Medical Correspondent

WASHINGTON, March 15 (UPI) -- The U.S. Department of Agriculture was quick to assure the public earlier this week that the third case of mad cow disease did not pose a risk to them, but what federal officials have not acknowledged is that this latest case indicates the deadly disease has been circulating in U.S. herds for at least a decade.

The second case, which was detected last year in a Texas cow and which USDA officials were reluctant to verify, was approximately 12 years old.

These two cases (the latest was detected in an Alabama cow) present a picture of the disease having been here for 10 years or so, since it is thought that cows usually contract the disease from contaminated feed they consume as calves. The concern is that humans can contract a fatal, incurable, brain-wasting illness from consuming beef products contaminated with the mad cow pathogen.

"The fact the Texas cow showed up fairly clearly implied the existence of other undetected cases," Dr. Paul Brown, former medical director of the National Institutes of Health's Laboratory for Central Nervous System Studies and an expert on mad cow-like diseases, told United Press International. "The question was, 'How many?' and we still can't answer that."

Brown, who is preparing a scientific paper based on the latest two mad cow cases to estimate the maximum number of infected cows that occurred in the United States, said he has "absolutely no confidence in USDA tests before one year ago" because of the agency's reluctance to retest the Texas cow that initially tested positive.

USDA officials finally retested the cow and confirmed it was infected seven months later, but only at the insistence of the agency's inspector general.

"Everything they did on the Texas cow makes everything they did before 2005 suspect," Brown said.

Despite this, Brown said the U.S. prevalence of mad cow, formally known as bovine spongiform encephalopathy, or BSE, did not significantly threaten human or cattle health.

"Overall, my view is BSE is highly unlikely to pose any important risk either in cattle feed or human feed," he said.

However, Jean Halloran of Consumers Union in Yonkers, N.Y., said consumers should be troubled by the USDA's secrecy and its apparent plan to dramatically cut back the number of mad cow tests it conducts.

"Consumers should be very concerned about how little we know about the USDA's surveillance program and the failure of the USDA to reveal really important details," Halloran told UPI. "Consumers have to be really concerned if they're going to cut back the program," she added.

Last year the USDA tested more than 300,000 animals for the disease, but it has proposed, even in light of a third case, scaling back the program to 40,000 tests annually.

"They seem to be, in terms of actions and policies, taking a lot more seriously the concerns of the cattle industry than the concerns of consumers," Halloran said. "It's really hard to know what it takes to get this administration to take action to protect the public."

The USDA has insisted that the safeguards of a ban on incorporating cow tissue into cattle feed (which is thought to spread the disease) and removal of the most infectious parts of cows, such as the brain and spinal cord, protect consumers. But the agency glosses over the fact that both of these systems have been revealed to be inadequately implemented.

The feed ban, which is enforced by the Food and Drug Administration, has been criticized by the Government Accountability Office in two reports, the most recent coming just last year. The GAO said the FDA's enforcement of the ban continues to have weaknesses that "undermine the nation's firewall against BSE."

USDA documents released last year showed more than 1,000 violations of the regulations requiring the removal of brains and spinal cords in at least 35 states, Puerto Rico and the Virgin Islands, with some plants being cited repeatedly for infractions. In addition, a violation of similar regulations that apply to beef exported to Japan is the reason why Japan closed its borders to U.S. beef in January six weeks after reopening them.

Other experts also question the adequacy of the USDA's surveillance system. The USDA insists the prevalence of mad cow disease is low, but the agency has provided few details of its surveillance program, making it difficult for outside experts to know if the agency's monitoring plan is sufficient.

"It's impossible to judge the adequacy of the surveillance system without having a breakdown of the tested population by age and risk status," Elizabeth Mumford, a veterinarian and BSE expert at Safe Food Solutions in Bern, Switzerland, a company that provides advice on reducing mad cow risk to industry and governments, told UPI.

"Everybody would be happier and more confident and in a sense it might be able to go away a little bit for (the USDA) if they would just publish a breakdown on the tests," Mumford added.

UPI requested detailed records about animals tested under the USDA's surveillance plan via the Freedom of Information Act in May 2004 but nearly two years later has not received any corresponding documents from the agency, despite a federal law requiring agencies to comply within 30 days. This leaves open the question of whether the USDA is withholding the information, does not have the information or is so haphazardly organized that it cannot locate it.

Mumford said the prevalence of the disease in U.S. herds is probably quite low, but there have probably been other cases that have so far gone undetected. "They're only finding a very small fraction of that low prevalence," she said.

Mumford expressed surprise at the lack of concern about the deadly disease from American consumers. "I would expect the U.S. public to be more concerned," she said.

Markus Moser, a molecular biologist and chief executive officer of Prionics, a Swiss firm that manufactures BSE test kits, told UPI one concern is that if people are infected, the mad cow pathogen could become "humanized" or more easily transmitted from person to person.

"Transmission would be much easier, through all kinds of medical procedures" and even through the blood supply, Moser said.

© Copyright 2006 United Press International, Inc. All Rights Reserved


http://www.upi.com/ConsumerHealthDaily/view.php?StoryID=20060315-055557-1284r



http://www.upi.com/Science_News/2003/12/30/Mad-Cow-Linked-to-thousands-of-CJD-cases/UPI-47861072816318/



CDC - Bovine Spongiform Encephalopathy and Variant Creutzfeldt ... Dr. Paul Brown is Senior Research Scientist in the Laboratory of Central Nervous System ... Address for correspondence: Paul Brown, Building 36, Room 4A-05, ...

http://www.cdc.gov/ncidod/eid/vol7no1/brown.htm




PAUL BROWN COMMENT TO ME ON THIS ISSUE

Tuesday, September 12, 2006 11:10 AM

"Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency." ........TSS


http://madcowtesting.blogspot.com/2009/07/mad-cow-cover-up-usa-masked-as-sporadic.html






OR, what the Honorable Phyllis Fong of the OIG found ;

Audit Report Animal and Plant Health Inspection Service Bovine Spongiform Encephalopathy (BSE) Surveillance Program ­ Phase II and Food Safety and Inspection Service

Controls Over BSE Sampling, Specified Risk Materials, and Advanced Meat Recovery Products - Phase III

Report No. 50601-10-KC January 2006

Finding 2 Inherent Challenges in Identifying and Testing High-Risk Cattle Still Remain


http://www.usda.gov/oig/webdocs/50601-10-KC.pdf





THIS is just ONE month report, of TWO recalls of prohibited banned MBM, which is illegal, mixed with 85% blood meal, which is still legal, but yet we know the TSE/BSE agent will transmit blood. we have this l-BSE in North America that is much more virulent and there is much concern with blood issue and l-BSE as there is with nvCJD in humans. some are even starting to be concerned with sporadic CJD and blood, and there are studies showing transmission there as well. ... this is one month recall page, where 10 MILLION POUNDS OF BANNED MAD COW FEED WENT OUT INTO COMMERCE, TO BE FED OUT. very little of the product that reaches commerce is ever returned via recall, very, very little. this was 2007, TEN YEARS AFTER THE AUGUST 4, 1997, PARTIAL AND VOLUNTARY MAD COW FEED BAN IN THE USA, that was nothing but ink on paper. i have listed the tonnage of mad cow feed that was in ALABAMA in one of the links too, this is where the infamous g-h-BSEalabama case was, a genetic relation matching the new sporadic CJD in the USA. seems this saga just keeps getting better and better.......$$$



10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN COMMERCE USA 2007

Date: March 21, 2007 at 2:27 pm PST

RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II

___________________________________

PRODUCT

Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007

CODE

Cattle feed delivered between 01/12/2007 and 01/26/2007

RECALLING FIRM/MANUFACTURER

Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.

Firm initiated recall is ongoing.

REASON

Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.

VOLUME OF PRODUCT IN COMMERCE

42,090 lbs.

DISTRIBUTION

WI

___________________________________

PRODUCT

Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007

CODE

The firm does not utilize a code - only shipping documentation with commodity and weights identified.

RECALLING FIRM/MANUFACTURER

Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007. Firm initiated recall is complete.

REASON

Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.

VOLUME OF PRODUCT IN COMMERCE

9,997,976 lbs.

DISTRIBUTION

ID and NV

END OF ENFORCEMENT REPORT FOR MARCH 21, 2007

http://www.fda.gov/Safety/Recalls/EnforcementReports/2007/ucm120446.htm



Tuesday, March 2, 2010

Animal Proteins Prohibited in Ruminant Feed/Adulterated/Misbranded Rangen Inc 2/11/10 USA

http://madcowfeed.blogspot.com/2010/03/animal-proteins-prohibited-in-ruminant.html



Monday, March 1, 2010

ANIMAL PROTEIN I.E. MAD COW FEED IN COMMERCE A REVIEW 2010

http://madcowfeed.blogspot.com/2010/03/animal-protien-ie-mad-cow-feed-in.html



Terry S. Singeltary Sr. (Submitted question): Monday, April 5, 2010

Update on Feed Enforcement Activities to Limit the Spread of BSE April 5, 2010

http://madcowfeed.blogspot.com/2010/04/update-on-feed-enforcement-activities.html



Friday, April 23, 2010

Upcoming BSE Webinar on Thursday, April 22, 2010 a review

http://bseusa.blogspot.com/2010/04/upcoming-bse-webinar-on-thursday-april.html



Suppressed peer review of Harvard study October 31, 2002.

October 31, 2002 Review of the Evaluation of the Potential for Bovine Spongiform Encephalopathy in the United States Conducted by the Harvard Center for Risk Analysis, Harvard School of Public Health and Center for Computational Epidemiology, College of Veterinary Medicine, Tuskegee University Final Report Prepared for U.S. Department of Agriculture Food Safety and Inspection Service Office of Public Health and Science Prepared by RTI Health, Social, and Economics Research Research Triangle Park, NC 27709 RTI Project Number 07182.024

http://www.fsis.usda.gov/oa/topics/BSE_Peer_Review.pdf



Sunday, February 14, 2010

[Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine Spongiform Encephalopathy (BSE)

http://bseusa.blogspot.com/2010/02/docket-no-fsis-2006-0011-fsis-harvard.html



PLEASE SEE FULL TEXT 98 PAGES HERE ;

[Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine Spongiform Encephalopathy (BSE)

http://www.fsis.usda.gov/OPPDE/Comments/2006-0011/2006-0011-1.pdf



Response to Public Comments on the Harvard Risk Assessment of Bovine Spongiform Encephalopathy

Update, October 31, 2005 INTRODUCTION The United States Department of Agriculture’s Food Safety and Inspection Service (FSIS) held a public meeting on July 25, 2006 in Washington, D.C. to present findings from the Harvard Risk Assessment of Bovine Spongiform Encephalopathy Update, October 31, 2005 (report and model located on the FSIS website: http://www.fsis.usda.gov/Science/Risk_Assessments/index.asp). Comments on technical aspects of the risk assessment were then submitted to FSIS. Comments were received from Food and Water Watch, Food Animal Concerns Trust (FACT), Farm Sanctuary, R-CALF USA, Linda A Detwiler, and Terry S. Singeltary. This document provides itemized replies to the public comments received on the 2005 updated Harvard BSE risk assessment. Please bear the following points in mind:

http://www.fsis.usda.gov/PDF/BSE_Risk_Assess_Response_Public_Comments.pdf



FDA STATEMENT FOR IMMEDIATE RELEASE May 4, 2004 Media Inquiries: 301-827-6242 Consumer Inquiries: 888-INFO-FDA

Statement on Texas Cow With Central Nervous System Symptoms

On Friday, April 30th, the Food and Drug Administration learned that a cow with central nervous system symptoms had been killed and shipped to a processor for rendering into animal protein for use in animal feed.

FDA, which is responsible for the safety of animal feed, immediately began an investigation. On Friday and throughout the weekend, FDA investigators inspected the slaughterhouse, the rendering facility, the farm where the animal came from, and the processor that initially received the cow from the slaughterhouse.

FDA's investigation showed that the animal in question had already been rendered into "meat and bone meal" (a type of protein animal feed). Over the weekend FDA was able to track down all the implicated material. That material is being held by the firm, which is cooperating fully with FDA.

Cattle with central nervous system symptoms are of particular interest because cattle with bovine spongiform encephalopathy or BSE, also known as "mad cow disease," can exhibit such symptoms. In this case, there is no way now to test for BSE. But even if the cow had BSE, FDA's animal feed rule would prohibit the feeding of its rendered protein to other ruminant animals (e.g., cows, goats, sheep, bison).

FDA is sending a letter to the firm summarizing its findings and informing the firm that FDA will not object to use of this material in swine feed only. If it is not used in swine feed, this material will be destroyed. Pigs have been shown not to be susceptible to BSE. If the firm agrees to use the material for swine feed only, FDA will track the material all the way through the supply chain from the processor to the farm to ensure that the feed is properly monitored and used only as feed for pigs.

To protect the U.S. against BSE, FDA works to keep certain mammalian protein out of animal feed for cattle and other ruminant animals. FDA established its animal feed rule in 1997 after the BSE epidemic in the U.K. showed that the disease spreads by feeding infected ruminant protein to cattle.

Under the current regulation, the material from this Texas cow is not allowed in feed for cattle or other ruminant animals. FDA's action specifying that the material go only into swine feed means also that it will not be fed to poultry.

FDA is committed to protecting the U.S. from BSE and collaborates closely with the U.S. Department of Agriculture on all BSE issues. The animal feed rule provides crucial protection against the spread of BSE, but it is only one of several such firewalls. FDA will soon be improving the animal feed rule, to make this strong system even stronger.

#

http://www.fda.gov/NewsEvents/Newsroom/PressAnnouncements/2004/ucm108292.htm



WHO WILL WATCH THE CHILDREN FOR CJD OVER THE NEXT 5 + DECADES ???

Do you actually believe that the USDA et al jumped in on the law suit against Westland/Hallmark, at the time the largest beef recall in USA history, just because a few animals were abused on a video, or to cover their ass, for letting our children, from school district to school district, from state to state, be fed dead stock downer cows.


>>> In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks... <<<


http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html


Do you actually believe all these schools recalled this meat because of a few cattle being abused, see list ; FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008


http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf


http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114


IF url does not work above, go to this link to find out if any of your children and their school were part of this recall ; go to this site ;


http://www.fns.usda.gov/fns/


left hand corner search ;


Hallmark/Westland Meat Packing Co. Beef Recall your should get this ;


http://65.216.150.153/texis/search?pr=FNS


1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ...


http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114


PLEASE SEE ALSO ;


Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.


http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf



PLEASE NOTE *


Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME.

snip...

The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...



http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf





PLEASE be aware, for 4 years, the USDA fed our children all across the Nation dead stock downer cows, the most high risk cattle for BSE aka mad cow disease and other dangerous pathogens. who will watch our children for CJD for the next 5+ decades ???

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE


http://downercattle.blogspot.com/2009/05/who-will-watch-children.html



http://downercattle.blogspot.com/




Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

page 114 ;

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf




International Society for Infectious Diseases Web: http://www.isid.org/

please see full text ;

http://transmissiblespongiformencephalopathy.blogspot.com/





To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.


http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2



please see full text ;

Wednesday, March 31, 2010

Atypical BSE in Cattle

http://bse-atypical.blogspot.com/2010/03/atypical-bse-in-cattle-position-post.html




CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER


>>> Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<


Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas

Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas.She left 6 Kids and a Husband.The Purpose of this web is to give information in Spanish to the Hispanic community, and to all the community who want's information about this terrible disease.-

Physician Discharge Summary, Parkland Hospital, Dallas Texas

Admit Date: 12/29/2009 Discharge Date: 1/20/2010 Attending Provider: Greenberg, Benjamin Morris; General Neurology Team: General Neurology Team

Linda was a Hispanic female with no past medical history presents with 14 months of incresing/progressive altered mental status, generalized weakness, inability to walk, loss of appetite, inability to speak, tremor and bowel/blader incontinence.She was, in her usual state of health up until February, 2009, when her husbans notes that she began forgetting things like names and short term memories. He also noticed mild/vague personality changes such as increased aggression. In March, she was involved in a hit and run MVA,although she was not injured. The police tracked her down and ticketed her. At that time, her son deployed to Iraq with the Army and her husband assumed her mentation changes were due to stress over these two events. Also in March, she began to have weakness in her legs, making it difficult to walk. Over the next few months, her mentation and personality changes worsened, getting to a point where she could no longer recognized her children. She was eating less and less. She was losing more weight. In the last 2-3 months, she reached the point where she could not walk without an assist, then 1 month ago, she stopped talking, only making grunting/aggressive sounds when anyone came near her. She also became both bowel and bladder incontinent, having to wear diapers. Her '"tremor'" and body jerks worsened and her hands assumed a sort of permanent grip position, leading her family to put tennis balls in her hands to protect her fingers.

The husband says that they have lived in Nebraska for the past 21 years. They had seen a doctor there during the summer time who prescribed her Seroquel and Lexapro, Thinking these were sx of a mood disorder. However, the medications did not help and she continued to deteriorate clinically. Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. The husband says that he does not know any fellow workers with a similar illness. He also says that she did not have any preceeding illness or travel.


http://www.recordandoalinda.com/index.php?option=com_content&view=article&id=19:cjd-english-info&catid=9:cjd-ingles&Itemid=8




let's take a closer look at this new prionpathy or prionopathy, and then let's look at the g-h-BSEalabama mad cow.

This new prionopathy in humans? the genetic makeup is IDENTICAL to the g-h-BSEalabama mad cow, the only _documented_ mad cow in the world to date like this, ......wait, it get's better. this new prionpathy is killing young and old humans, with LONG DURATION from onset of symptoms to death, and the symptoms are very similar to nvCJD victims, OH, and the plaques are very similar in some cases too, bbbut, it's not related to the g-h-BSEalabama cow, WAIT NOW, it gets even better, the new human prionpathy that they claim is a genetic TSE, has no relation to any gene mutation in that family. daaa, ya think it could be related to that mad cow with the same genetic make-up ??? there were literally tons and tons of banned mad cow protein in Alabama in commerce, and none of it transmitted to cows, and the cows to humans there from ??? r i g h t $$$

ALABAMA MAD COW g-h-BSEalabama

In this study, we identified a novel mutation in the bovine prion protein gene (Prnp), called E211K, of a confirmed BSE positive cow from Alabama, United States of America. This mutation is identical to the E200K pathogenic mutation found in humans with a genetic form of CJD. This finding represents the first report of a confirmed case of BSE with a potential pathogenic mutation within the bovine Prnp gene. We hypothesize that the bovine Prnp E211K mutation most likely has caused BSE in "the approximately 10-year-old cow" carrying the E221K mutation.


http://www.plospathogens.org/article/info%3Adoi%2F10.1371%2Fjournal.ppat.1000156



http://www.plospathogens.org/article/fetchObjectAttachment.action?uri=info%3Adoi%2F10.1371%2Fjournal.ppat.1000156&representation=PDF




Saturday, August 14, 2010

BSE Case Associated with Prion Protein Gene Mutation (g-h-BSEalabama) and VPSPr PRIONPATHY


(see mad cow feed in COMMERCE IN ALABAMA...TSS)


http://prionpathy.blogspot.com/2010/08/bse-case-associated-with-prion-protein.html




2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html



Monday, October 19, 2009

Atypical BSE, BSE, and other human and animal TSE in North America Update October 2009

http://bse-atypical.blogspot.com/2009/10/atypical-bse-bse-and-other-human-and.html



Tuesday, September 14, 2010

Feed Safety and BSE/Ruminant Feed Ban Support Project (U18)

http://madcowfeed.blogspot.com/2010/09/feed-safety-and-bseruminant-feed-ban.html



Tuesday, August 03, 2010

Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein

http://creutzfeldt-jakob-disease.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html



Monday, August 9, 2010

Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein or just more PRIONBALONEY ?

http://prionunitusaupdate2008.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html



***

Thursday, July 10, 2008

A Novel Human Disease with Abnormal Prion Protein Sensitive to Protease update July 10, 2008 Friday, June 20, 2008

http://cjdmadcowbaseoct2007.blogspot.com/2008/07/novel-human-disease-with-abnormal-prion.html




P.9.21

Molecular characterization of BSE in Canada

Jianmin Yang1, Sandor Dudas2, Catherine Graham2, Markus Czub3, Tim McAllister1, Stefanie Czub1 1Agriculture and Agri-Food Canada Research Centre, Canada; 2National and OIE BSE Reference Laboratory, Canada; 3University of Calgary, Canada

Background: Three BSE types (classical and two atypical) have been identified on the basis of molecular characteristics of the misfolded protein associated with the disease. To date, each of these three types have been detected in Canadian cattle.

Objectives: This study was conducted to further characterize the 16 Canadian BSE cases based on the biochemical properties of there associated PrPres. Methods: Immuno-reactivity, molecular weight, glycoform profiles and relative proteinase K sensitivity of the PrPres from each of the 16 confirmed Canadian BSE cases was determined using modified Western blot analysis.

Results: Fourteen of the 16 Canadian BSE cases were C type, 1 was H type and 1 was L type. The Canadian H and L-type BSE cases exhibited size shifts and changes in glycosylation similar to other atypical BSE cases. PK digestion under mild and stringent conditions revealed a reduced protease resistance of the atypical cases compared to the C-type cases. N terminal- specific antibodies bound to PrPres from H type but not from C or L type. The C-terminal-specific antibodies resulted in a shift in the glycoform profile and detected a fourth band in the Canadian H-type BSE.

Discussion: The C, L and H type BSE cases in Canada exhibit molecular characteristics similar to those described for classical and atypical BSE cases from Europe and Japan. This supports the theory that the importation of BSE contaminated feedstuff is the source of C-type BSE in Canada. It also suggests a similar cause or source for atypical BSE in these countries.

http://www.prion2009.com/sites/default/files/Prion2009_Book_of_Abstracts.pdf



Wednesday, August 11, 2010

REPORT ON THE INVESTIGATION OF THE SIXTEENTH CASE OF BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN CANADA

http://bse-atypical.blogspot.com/2010/08/report-on-investigation-of-sixteenth.html


Thursday, August 19, 2010


REPORT ON THE INVESTIGATION OF THE SEVENTEENTH CASE OF BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN CANADA

http://bseusa.blogspot.com/2010/08/report-on-investigation-of-seventeenth.html



Thursday, August 19, 2010

SCRAPIE CANADA UPDATE Current as of 2010-07-31 The following table lists sheep flocks and/or goat herds confirmed to be infected with scrapie in Canada in 2010.

Current as of: 2010-07-31

http://nor-98.blogspot.com/2010/08/scrapie-canada-update-current-as-of.html



Wednesday, August 18, 2010

Incidence of CJD Deaths Reported by CJD-SS in Canada as of July 31, 2010

http://creutzfeldt-jakob-disease.blogspot.com/2010/08/incidence-of-cjd-deaths-reported-by-cjd.html



Monday, August 9, 2010

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)

(please watch and listen to the video and the scientist speaking about atypical BSE and sporadic CJD and listen to Professor Aguzzi)

http://prionunitusaupdate2008.blogspot.com/2010/08/national-prion-disease-pathology.html



Tuesday, September 14, 2010

Transmissible Spongiform Encephalopathies Advisory Committee; Notice of Meeting October 28 and 29, 2010 (COMMENT SUBMISSION)

http://tseac.blogspot.com/2010/09/transmissible-spongiform_14.html



Monday, September 13, 2010

atypical BSE strains and sporadic CJD strains, is there a connection and why shouldn't there be $

http://bse-atypical.blogspot.com/2010/09/atypical-bse-strains-and-sporadic-cjd.html



Wednesday, September 08, 2010

Emerging Infectious Diseases: CJD, BSE, SCRAPIE, CWD, PRION, TSE Evaluation to Implementation for Transfusion and Transplantation September 2010

http://vcjdtransfusion.blogspot.com/2010/09/emerging-infectious-diseases-cjd-bse.html



Wednesday, September 08, 2010

CWD PRION CONGRESS SEPTEMBER 8-11 2010

http://chronic-wasting-disease.blogspot.com/2010/09/cwd-prion-2010.html



Friday, August 27, 2010

NEW ATYPICAL NOR-98 SCRAPIE CASE DETECTED IDAHO NOW 5 CASES DOCUMENTED 2010

http://nor-98.blogspot.com/2010/08/new-atypical-nor-98-scrapie-case.html



Sunday, April 18, 2010

SCRAPIE AND ATYPICAL SCRAPIE TRANSMISSION STUDIES A REVIEW 2010

http://scrapie-usa.blogspot.com/2010/04/scrapie-and-atypical-scrapie.html



++++++++++++++++ ************************** ================


Tuesday, July 13, 2010

(SEE BEEF PRODUCTS EXPORTED TO AUSTRALIA FROM USA...TSS)

AUSTRALIAN QUESTIONNAIRE TO ASSESS BSE RISK (OIE) Terrestrial Animal Health Code, 2009 and USA export risk factor for BSE to Australia

http://usdameatexport.blogspot.com/2010/07/australian-questionnaire-to-assess-bse.html



Saturday, August 14, 2010

USA NON-SPECIES CODING SYSTEM (BEEF IMPORT EXPORT BSE RISK THERE FROM)

US denies it's illegally sending beef to Australia ?

Friday, 13/08/2010

http://usdameatexport.blogspot.com/2010/08/usa-non-species-coding-system-beef.html



Saturday, June 19, 2010

U.S. DENIED UPGRADED BSE STATUS FROM OIE

http://usdameatexport.blogspot.com/2010/06/us-denied-upgraded-bse-status-from-oie.html



Sunday, August 15, 2010

ATYPICAL BSE NOW LINKED TO CAUSING SPORADIC CJD OVERSEAS Commonwealth of Australia

http://bse-atypical.blogspot.com/2010/08/atypical-bse-now-linked-to-causing.html





Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518

Friday, September 10, 2010

Austria has been hit by the eighth case of mad-cow disease (BSE) this year

10. 09. 10. - 12:00

Dead cow had BSE, test shows Austria has been hit by the eighth case of mad-cow disease (BSE) this year.

The federal health ministry announced today (Fri) post mortem tests have shown that a 15-year-old cow which died of old age on a farm in the Lower Austrian district of Wiener Neustadt had the disease.

The ministry, headed by Social Democrat (SPÖ) Alois Stöger, stressed customers’ health had not been endangered since none of the animal's meat had gone on sale.

Meanwhile, People’s Party (ÖVP) Environment Minister Nikolaus Berlakovich unveiled plans to set up a country-wide egg-farming database.

Berlakovich’s initiative comes after it emerged that a Styrian firm imported substandard eggs from Poland and Hungary to sell them as local products.

The minister said he thought of the already existing anti-BSE database as an example for the planned egg-trading platform.

Berlakovich said all traders and sellers of eggs will have to inform authorities about their business activities by entering sale figures and other information in the database.

http://austrianindependent.com/news/General_News/2010-09-10/4529/Dead_cow_had_BSE,_test_shows


Pressemeldungen 09.09.2010 15:37 Gesundheitsministerium bestätigt 8. BSE-Fall in Österreich Wien (BGF) - Am Dienstag, den 7. September 2010, wurde ein 1995 geborenes Rind aus Niederösterreich im Zuge der Untersuchung von sogenannten Risikotieren im Rahmen eines Schnelltests positiv auf BSE getestet. Das Untersuchungsergebnis wurde heute, Donnerstag, den 9. September, durch das nationale Referenzlabor der Österreichischen Agentur für Gesundheit und Ernährungssicherheit (AGES) in Mödling endgültig bestätigt. Da das Tier bereits verendet war und eine Untersuchung in der Tierkörperverwertung veranlasst wurde, bestand keine Gefahr, dass Fleisch in die Lebensmittelkette gelangte. Es handelt sich um den insgesamt achten BSE-Fall in Österreich.

Die Veterinärbehörde Niederösterreich hat in Abstimmung mit dem Bundesministerium für Gesundheit unverzüglich alle notwendigen Vorkehrungen getroffen. "Durch die gute Zusammenarbeit zwischen den Bundes- und Landesbehörden, bestand nie ein Risiko für die Konsumentinnen und Konsumenten. Das zeigt auch, dass die Maßnahmen zur Bekämpfung von BSE und zur Absicherung des Konsumentenschutzes greifen", betonte Gesundheitsminister Alois Stöger. Weitere Informationen zum Thema BSE: http://www.bmg.gv.at Rückfragehinweis: Bundesministerium für Gesundheit Fabian Fußeis, Pressesprecher Radetzkystraße 2, 1030 Wien Tel.: +43/1/71100-4505 , Fax: +43/1/71100-14304 mailto:fabian.fusseis@bmg.gv.at http://www.bmg.gv.at Digitale Pressemappe: http://www.ots.at/pressemappe/52/aom *** OTS-ORIGINALTEXT PRESSEAUSSENDUNG UNTER AUSSCHLIESSLICHER INHALTLICHER VERANTWORTUNG DES AUSSENDERS - WWW.OTS.AT *** OTS0284 2010-09-09/15:37

http://www.bmg.gv.at/cms/site/presse_detail.html?channel=CH0616&doc=CMS1284039658617


News Am Dienstag, den 7. September 2010, wurde ein 1995 geborenes Rind aus Niederösterreich im Zuge der Untersuchung von sogenannten Risikotieren im Rahmen eines Schnelltests positiv auf BSE getestet. Das Untersuchungsergebnis wurde am Donnerstag, den 09. September, durch das nationale Referenzlabor der Österreichischen Agentur für Gesundheit und Ernährungssicherheit (AGES) in Mödling endgültig bestätigt. Da das Tier bereits verendet war und eine Untersuchung in der Tierkörperverwertung veranlasst wurde, bestand keine Gefahr, dass Fleisch in die Lebensmittelkette gelangte. Es handelt sich um den insgesamt achten BSE-Fall in Österreich. Die Veterinärbehörde Niederösterreich hat in Abstimmung mit dem Bundesministerium für Gesundheit unverzüglich alle notwendigen Vorkehrungen getroffen. „Durch die gute Zusammenarbeit zwischen den Bundes- und Landesbehörden, bestand nie ein Risiko für die Konsumentinnen und Konsumenten. Das zeigt auch, dass die Maßnahmen zur Bekämpfung von BSE und zur Absicherung des Konsumentenschutzes greifen“, betonte Gesundheitsminister Alois Stöger.

Aktuelle Informationen BSE (DOC 42 KB) 9.09.2010 - 8. Fall bestätigt

http://www.bmg.gv.at/cms/site/news_detail.html?channel=CH0518&doc=CMS1284038655071



============================


Friday, September 3, 2010

Dutch report positive test for mad cow disease

http://madcowtesting.blogspot.com/2010/09/dutch-report-positive-test-for-mad-cow.html



TSS

Friday, September 3, 2010

Dutch report positive test for mad cow disease

Subject: Dutch report positive test for mad cow disease

Dutch report positive test for mad cow disease

AMSTERDAM, Sept 3 Fri Sep 3, 2010 7:00am EDT

AMSTERDAM, Sept 3 (Reuters) - A 10-year-old cow in the Netherlands has tested positive for BSE, more commonly known as "mad cow" disease, the first such result in more than two years, the Dutch government said on Friday.

The government ministry responsible for food quality said the animal tested positive for the brain-wasting disease bovine spongiform encephalopathy at a slaughterhouse.

It was the first positive test for BSE in the country since May 2008, the ministry said in a statement.

A spokesman for the ministry told Reuters the cow's meat was withdrawn from the food chain after a first positive test, while a second test confirmed the result.

All cows sent to slaughter in the country are tested and held aside for the results before their meat enters the system.

Mad cow disease is of particular concern because it has been known to cause a related brain-wasting disease in humans who have eaten contaminated meat.

Three people have died in the Netherlands from Creutzfeldt-Jakob disease after eating meat from a BSE positive cow. The last reported death was in January 2009. (Reporting by Ben Berkowitz; editing by James Jukwey)

http://www.reuters.com/article/idUSLDE6820VK20100903


Na twee jaar weer BSE-koe aangetroffen Persbericht 03-09-2010

Er is een geval van BSE vastgesteld. Het gaat om een ruim tien jaar oude koe die op het slachthuis is getest. Dit past in de verwachting van het Centraal Veterinair Instituut in Lelystad dat Nederland in de komende jaren af en toe een besmette koe zal tegenkomen.

Het ministerie van LNV is blij dat Nederland ondanks een zeer intensief testprogramma gedurende een periode van ruim twee jaar geen enkel geval van BSE aantrof. Het laatste geval was in mei 2008. In 2009 testte Nederland 405.000 runderen op BSE.

http://www.minlnv.nl/portal/page?_pageid=116,1640333&_dad=portal&_schema=PORTAL&p_news_item_id=2007820


BSE For some years now the Netherlands has been taking measures to prevent BSE (Bovine Spongiforme Encephalopathy) in its cattle herd. All EU measures were implemented, though the Netherlands has also anticipated European regulation and introduced additional measures of its own.

In spite of all these efforts the Netherlands has not been free of BSE. It was first diagnosed in the Netherlands in 1997.

Measures taken by the Dutch Government Over the years a broad package of measures has been built up to combat BSE. This is partly aimed at food safety, partly at the eradication of BSE. These measures follow the recommendations of the Office International des Epizoties (OIE), and Decisions of the EU.

The measures involve:

Tracking down diseased or suspect cattle. Since 1989 it is been compulsory for owners and veterinarians to report any cattle that show symptoms of BSE to the authorities. Evaluation of the animal's health at the slaughterhouse, prior to slaughter. Compulsory removal of risk material on slaughter; This measure was introduced in the Netherlands in 1997. It has applied to all European Member States since 1 October 2000. Treatment of animal by-products used in animal feed at 133°C and 3 bar during 20 min. since the seventies. A ban on the use of animal protein in animal feed for domestic farm animals (such as cattle). Testing for BSE on all slaughtered cattle older than 30 months. Further information on the most important measures is given below.

Removal of risk material The most important measure taken to protect the consumer against BSE is the decision that so-called risk material must be removed in the slaughterhouse. The disease-causing prions do not occur in the whole animal. They are concentrated in the brains, spinal cord and some other risk material. This material is removed on slaughter and incinerated, and so eliminated from the food chain. Disease-causing organisms have never been found in meat taken from cattle muscle (steak, etc.). The removal of risk material has been compulsory since 1997.

Ban on animal protein The aim is to eliminate BSEby removing the most important source of infection (infected animal protein).

Since 1989 there has been a ban in the Netherlands on the use of remains of ruminants in ruminant feed (cattle, sheep and goats). This ban has been tightened on a number of occasions. Since 1994 no animal protein originating from mammals (previously ruminants) may be used in ruminant feed. Since 1999 the production of feed for ruminants and feed for non-ruminants containing animal protein is totally separated. This measures prevents any contamination of feed for ruminants with animal protein. Since 1 January 2001 feed containing animal protein from mammals is not only banned for ruminants, but also for all domestic farm animals, such as pigs and chickens. Compulsory BSE test From 1 January 2001 all cattle older than 30 months presented for slaughter are subjected to a rapid BSE test, approved by the European Commission. In order to carry out these tests a piece of brain tissue is removed from the cattle. If the result is positive the final diagnosis is made by traditional microscopic study of brain tissue, according to OIE.

In addition to the testing of cattle older than 30 months, risk material is removed from slaughtered cattle intended for human consumption.

http://www.minlnv.nl/portal/page?_pageid=116,1640387&_dad=portal&_schema=PORTAL&p_document_id=111059&p_node_id=5550285&p_mode=


http://www.minlnv.nl/portal/page?_pageid=116,1640440&_dad=portal&_schema=PORTAL&p_node_id=8887325



J Neurol. 2007 July; 254(7): 958–960. Published online 2007 April 21. doi: 10.1007/s00415-006-0360-3. PMCID: PMC2779429

Copyright © Steinkopff-Verlag 2007

The first case of variant Creutzfeldt-Jakob disease in the Netherlands


http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2779429/



Eurosurveillance, Volume 11, Issue 26, 29 June 2006 Articles C van Duijn1, H Ruijs2, A Timen2

--------------------------------------------------------------------------------

Citation style for this article: van Duijn C, Ruijs H, Timen A.


Second probable case of vCJD in the Netherlands.


Euro Surveill. 2006;11(26):pii=2991.

Available online: http://www.eurosurveillance.org/ViewArticle.aspx?ArticleId=2991



Date of submission:

Second probable case of vCJD in the Netherlands


http://www.eurosurveillance.org/ViewArticle.aspx?ArticleId=2991


TO DATE, 3 CASES OF nvCJD have been documented in the Netherlands ;

vCJD cases Worldwide (Netherlands = 3 cases nvCJD documented to date August 2010)

Country 1995 1996 1997 1998 1999 2000 2001 2002 2003 2004 2005 2006 2007 2008 2009 2010 Alive Total

Netherlands 1 1 1 3

http://www.eurocjd.ed.ac.uk/surveillance%20data%204.htm


Total Cases of Sporadic CJD (Deaths)

Sporadic CJD: Definite and probable cases

Country 1993 1994 1995 1996 1997 1998 1999 2000 2001 2002 2003 2004 2005 2006 2007 2008 2009 Total

Netherlands 12 18 8 14 18 17 19 10 14 18 12 20 20 22 15 16 11 264

http://www.eurocjd.ed.ac.uk/surveillance%20data%203.htm


BSE CJD NORTH AMERICA 2010

let's take a closer look at this new prionpathy or prionopathy, and then let's look at the g-h-BSEalabama mad cow.

This new prionopathy in humans? the genetic makeup is IDENTICAL to the g-h-BSEalabama mad cow, the only _documented_ mad cow in the world to date like this, ......wait, it get's better. this new prionpathy is killing young and old humans, with LONG DURATION from onset of symptoms to death, and the symptoms are very similar to nvCJD victims, OH, and the plaques are very similar in some cases too, bbbut, it's not related to the g-h-BSEalabama cow, WAIT NOW, it gets even better, the new human prionpathy that they claim is a genetic TSE, has no relation to any gene mutation in that family. daaa, ya think it could be related to that mad cow with the same genetic make-up ??? there were literally tons and tons of banned mad cow protein in Alabama in commerce, and none of it transmitted to cows, and the cows to humans there from ??? r i g h t $$$

ALABAMA MAD COW g-h-BSEalabama

In this study, we identified a novel mutation in the bovine prion protein gene (Prnp), called E211K, of a confirmed BSE positive cow from Alabama, United States of America. This mutation is identical to the E200K pathogenic mutation found in humans with a genetic form of CJD. This finding represents the first report of a confirmed case of BSE with a potential pathogenic mutation within the bovine Prnp gene. We hypothesize that the bovine Prnp E211K mutation most likely has caused BSE in "the approximately 10-year-old cow" carrying the E221K mutation.

http://www.plospathogens.org/article/info%3Adoi%2F10.1371%2Fjournal.ppat.1000156


http://www.plospathogens.org/article/fetchObjectAttachment.action?uri=info%3Adoi%2F10.1371%2Fjournal.ppat.1000156&representation=PDF


Saturday, August 14, 2010

BSE Case Associated with Prion Protein Gene Mutation (g-h-BSEalabama) and VPSPr PRIONPATHY

(see mad cow feed in COMMERCE IN ALABAMA...TSS)

http://prionpathy.blogspot.com/2010/08/bse-case-associated-with-prion-protein.html


Wednesday, July 28, 2010

re-Freedom of Information Act Project Number 3625-32000-086-05, Study of Atypical BSE UPDATE July 28, 2010

http://bse-atypical.blogspot.com/2010/07/re-freedom-of-information-act-project.html


Tuesday, August 03, 2010

Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein

http://creutzfeldt-jakob-disease.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html


Monday, August 9, 2010

Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein or just more PRIONBALONEY ?

http://prionunitusaupdate2008.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html


Sunday, August 29, 2010

Prion Disease Round Table Conducted Wednesday December 11, 2003 at Denver, Colorado R-CALF-USA Sponsored (REVISITED AUGUST 2010)

http://bseusa.blogspot.com/2010/08/prion-disease-round-table-conducted.html


Friday, August 27, 2010

NEW ATYPICAL NOR-98 SCRAPIE CASE DETECTED IDAHO NOW 5 CASES DOCUMENTED 2010

http://nor-98.blogspot.com/2010/08/new-atypical-nor-98-scrapie-case.html


Thursday, August 19, 2010

REPORT ON THE INVESTIGATION OF THE SEVENTEENTH CASE OF BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN CANADA

http://bseusa.blogspot.com/2010/08/report-on-investigation-of-seventeenth.html


Thursday, August 19, 2010

SCRAPIE CANADA UPDATE Current as of 2010-07-31 The following table lists sheep flocks and/or goat herds confirmed to be infected with scrapie in Canada in 2010.

Current as of: 2010-07-31

http://nor-98.blogspot.com/2010/08/scrapie-canada-update-current-as-of.html


14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf


To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.

http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2


Wednesday, March 31, 2010

Atypical BSE in Cattle

http://bse-atypical.blogspot.com/2010/03/atypical-bse-in-cattle-position-post.html


Friday, May 14, 2010

Prion Strain Mutation Determined by Prion Protein Conformational Compatibility and Primary Structure

Published Online May 13, 2010 Science DOI: 10.1126/science.1187107 Science Express Index

http://chronic-wasting-disease.blogspot.com/2010/05/prion-strain-mutation-determined-by.html


Thursday, June 03, 2010

Prion Strain Mutation and Selection John Collinge MEDICINE

http://chronic-wasting-disease.blogspot.com/2010/06/prion-strain-mutation-and-selection.html


Wednesday, August 18, 2010

Incidence of CJD Deaths Reported by CJD-SS in Canada as of July 31, 2010

http://creutzfeldt-jakob-disease.blogspot.com/2010/08/incidence-of-cjd-deaths-reported-by-cjd.html


Monday, August 9, 2010

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)

(please watch and listen to the video and the scientist speaking about atypical BSE and sporadic CJD and listen to Professor Aguzzi)

http://prionunitusaupdate2008.blogspot.com/2010/08/national-prion-disease-pathology.html


CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER


>>>Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<


Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas

Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas.She left 6 Kids and a Husband.The Purpose of this web is to give information in Spanish to the Hispanic community, and to all the community who want's information about this terrible disease.-

Physician Discharge Summary, Parkland Hospital, Dallas Texas

Admit Date: 12/29/2009 Discharge Date: 1/20/2010 Attending Provider: Greenberg, Benjamin Morris; General Neurology Team: General Neurology Team

Linda was a Hispanic female with no past medical history presents with 14 months of incresing/progressive altered mental status, generalized weakness, inability to walk, loss of appetite, inability to speak, tremor and bowel/blader incontinence.She was, in her usual state of health up until February, 2009, when her husbans notes that she began forgetting things like names and short term memories. He also noticed mild/vague personality changes such as increased aggression. In March, she was involved in a hit and run MVA,although she was not injured. The police tracked her down and ticketed her. At that time, her son deployed to Iraq with the Army and her husband assumed her mentation changes were due to stress over these two events. Also in March, she began to have weakness in her legs, making it difficult to walk. Over the next few months, her mentation and personality changes worsened, getting to a point where she could no longer recognized her children. She was eating less and less. She was losing more weight. In the last 2-3 months, she reached the point where she could not walk without an assist, then 1 month ago, she stopped talking, only making grunting/aggressive sounds when anyone came near her. She also became both bowel and bladder incontinent, having to wear diapers. Her '"tremor'" and body jerks worsened and her hands assumed a sort of permanent grip position, leading her family to put tennis balls in her hands to protect her fingers.

The husband says that they have lived in Nebraska for the past 21 years. They had seen a doctor there during the summer time who prescribed her Seroquel and Lexapro, Thinking these were sx of a mood disorder. However, the medications did not help and she continued to deteriorate clinically. Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. The husband says that he does not know any fellow workers with a similar illness. He also says that she did not have any preceeding illness or travel.

http://www.recordandoalinda.com/index.php?option=com_content&view=article&id=19:cjd-english-info&catid=9:cjd-ingles&Itemid=8


>>>Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<


Monday, March 29, 2010

Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas

http://creutzfeldt-jakob-disease.blogspot.com/2010/03/irma-linda-andablo-cjd-victim-she-died.html


CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER

http://cjdtexas.blogspot.com/2010/03/cjd-texas-38-year-old-female-worked.html


Thursday, August 12, 2010

USA Blood products, collected from a donor who was at risk for vCJD, were distributed July-August 2010

http://creutzfeldt-jakob-disease.blogspot.com/2010/08/usa-blood-products-collected-from-donor.html


Thursday, August 12, 2010

Seven main threats for the future linked to prions

http://prionpathy.blogspot.com/2010/08/seven-main-threats-for-future-linked-to.html


http://prionpathy.blogspot.com/



TSS